It is possible that DES promotes a transgenerational phenotype
Prenatal exposure to DES may lead to an altered immune status
Reproductive Dysfunction in Daughters and Sons
Defining a DES “programming window
DES adverse impact on fetal germ cells, impairing neurodevelopment of offspring
DES imparts a permanent estrogen imprint
DES exposure induces changes in the expression of many genes such as Wnt7a, Hoxa9, Hoxa10, Hoxa11, lactoferrin and c-fos genes
Proceedings of the Summit on Environmental Challenges to Reproductive Health and Fertility: Executive Summary
DES silencing of KLF9 may contribute to the etiology of endometrial cancers initiated by aberrant ESR1 signaling
Vascular density, integrity of vasculatures, and expression of angiogenic factors and their receptors in the uteri treated with DES
